This is a long post. It details the molecular reality of the origin of the FauciFlu. This man should be in jail, not collecting billions!!

Here is video of money trail: https://thedonald.win/p/11R4X9yoYV/fauci-is-soooooo-busted-on-covid/c/

As a gene jock, here is my analysis of the forensic molecular biology to go along with the vid:

The FauciFlu is 100% manufactured.

Here is how it was done in broad strokes and there are genetic fingerprints that verifies this.

Step 1: Start with a template naturally occurring virus. Probably Coronavirus RTG13 which shares 96% homology to Coronavirus 19. (I.e. 96% of the 30k nucleotides are the same)

See this paper for some background: http://www.barc.gov.in/publications/nl/2020/2020091003.pdf

Now, the RTG13 was collected in 2013 (hence the 13 in the name) from an abandoned mine about 1000miles from Wuhan and taken to Wuhan for study by Shi Zhengli, PhD, known as the “bat woman” because she studies bats and bat viruses. Note that Wuhan local scientists have said that this bat is NOT sold in the Wuhan market for food....the bats live 1000 miles away from Wuhan! Not practical to be sold in the market and people that reportedly go to the market confirm they are not sold there. Covid19 did NOT come from that market spontaneously. Perhaps the final manipulated virus got leaked there by a lab worker...it is very close to Shi’s lab, I.e. close enough for workers to go there for lunch or to buy groceries after work. Literally only blocks away.

Step 2: Do gain of function manipulations on the RTG13 virus to make it infect human cells.

There were two parts (at least) to this. The goal as noted in the documentary is to increase the infectivity into human cells. The RTG13 virus is NOT that dangerous to humans because it does not infect human cells that well. At first Shi would not have known the reasons why it wasn’t infective to human cells specifically. As molecular biologist she would have guessed that a virus that causes disease in humans would express a protein at its surface that binds to a protein in a human cell which leads to the virus getting taken up by the human cell where it can hijack the cell translation/transcription machinery to make thousands of virus copies. These copies are released to infect neighboring cells in an exponential reaction that leads to the infection. She would know that she would have to somehow manipulate the viral protein(s) of RTG13 so it would do that.

But, at the beginning, she probably would not know exactly which protein on the virus was critical and how exactly it needed to be altered to make the binding to the cell membrane more effective.

I guess I should note that there was a fair amount of information about SARS-Cov1 by 2010 so she probably did know a bit about the virus “spike” protein, which is the key protein on the RTG13 virus surface to bind to the cell membrane. I would have to look back through the SARS-Cov1 research...they may have even known that the spike protein binds to the cell Ace2 protein. Anyway, I digress because the key is that RTG13 did not infect human cells well and they did not know specifically how to manipulate it to increase that infectivity. They would have to use random mutagenesis.

They needed to do gain of function manipulation with directed evolution to achieve this:

Already in 2015 she has figured out quite a bit about the spike protein, Ace2 and manipulating it to “improve” infectivity. See her paper detailing this:

https://scholar.google.com/scholar?start=10&q=Zhengli+and+virus&hl=en&as_sdt=0,5#d=gs_qabs&u=%23p%3D07KsBYcZKcEJ

(Btw- this is actually a pretty relevant paper about coronavirus treatment and vaccine. Strong data about poor response to attenuated vaccine hence the mRNA vaccine currently being rolled out. That is another discussion so I will not focus on the vaccine any further here)

In this next paper she shows that a second step is required after binding of the viral spike protein to the Ace2 protein. This step is for the spike protein to be cleaved by proteases on the cell membrane.

https://scholar.google.com/scholar?hl=en&as_sdt=0%2C5&q=Zhengli+and+virus+and+spike&btnG=#d=gs_qabs&u=%23p%3DTefot1GEWGoJ

So these are the two parts to achieve human transmission. 1. Improve binding of the RTG13 to human Ace2 and 2. Improve cleavage of the viral spike protein by human protease. Achieve these two steps and you have successfully changed RTF13 into a human viral pathogen...a human viral pathogen that we call Covid19.

LAB METHODS - Part 1: Directed evolution to improve binding (but leaves a finger print).

The goal of this step was to improve binding of the RTG13 virus to human Ace2. This is done by growing up a bunch of virus but doing it in the presence of an inhibitor of transcription fidelity. This is relatively easy, there are well established protocols using 5-FU to inhibit the ExoN enzyme responsible for transcription integrity.

See description here: https://www.nature.com/articles/nrmicro3125

However, as noted in this paper the induced errors have a specific frequency. Not all errors are equally likely.

THIS IS THE MUTATION FREQUENCY FINGERPRINT PRIVES MANIPULATION by Shi’s lab rather than it occurring spontaneously in nature.

Let me say that another way. Coronavirus19 and RTG13 have genetic sequences that are 96% similar. The 4% that are different follow this frequency of changes. This is a fingerprint that it occurred in a test tube. Would not have occurred naturally.

So, to do this, you create literally trillions and trillions of randomly mutated viruses, each one with a different set of random mutations, by growing them up in the presence of 5-FU. But how do you isolate the very few virus particles amongst all those trillions that happen to have randomly mutated to bind better to human Ace2 protein? Pretty standard protocol actually: you take a bunch of purified human Ace2 and attach it to tiny glass beads. Then you combine the glass beads and the mutated virus stew (the virus is still in solution) and let any random mutants that happen to bind to Ace2 do that. Next wash out the stew (including trillions of rand mutants whose mutations did not promote Ace2 binding) and rinse the beads. This gets rid of the virus that did not bind to the Ace2 on the beads (I.e. use a centrifuge to spin the beads down with each wash). Next use a mild denaturant to release the virus mutants that DID bind to the Ace2 on the beads. Grow this virus up and possibly repeat the cycle a couple of times. Pretty soon you have randomly mutated virus that binds very well to human Ace2.

It also has a mutation frequency that is a fingerprint for lab manipulation!

(Btw- Shi published an article in the 2014 time frame detailing this very protocol. Can’t find the ref just now but I have seen it. She knows how to do this step)

Part 2: So, the virus now binds to human ACE2 very well but it still does not replicate efficiently so does not cause disease because the spike protein needs to be cleaved by HUMAN furin proteases.

The next step is to insert a known Furin cleavage consensus site into the viral genome. This is also standard molecular technology probably using PCR insertion also mutagenesis, a protocol she describes it in the above paper so she is capable of this as well.

This also leaves a fingerprint of sorts because it turns out the furin cleavage site she inserted is not found in any other coronavirus or other related virus (read in civit if you are a gene jock or virologist).

There you have it. Production of a pathogenic coronavirus from the RTG13 template virus with two genetic fingerprints of the manipulation. Could not have occurred “naturally”.

Of note, there was a video (probably deleted now) where someone (who speaks Chinese) noted that a job posting was placed on Shi’a lab website in 10/2019 at the Wuhan Virology website stating Shi was looking for a new grad student because she had just made an important discovery. This video journalist also noted that the grad student went missing in 12/2019 and many in China view this young grad student as patient zero. Perhaps she seeded the market?? Shi’s lab was noted to have poor safety protocols. I believe that Obama actually went there to eval the safety because many in the virology research community were distressed about this gain of function research at that time. They realized the danger. Fauci fiercely defended it and, as noted in the above documentary, actually funded it with American tax dollars.

I suspect that Shi’a lab made the second step in 10/2019 adding the furin cleavage site but that the virus was either accidentally released by this grad student or was released on purpose by the CCP.

Again, as the documentary details, Fauci funded this. It is the Fuaci flu!