Even if you do have heart issues, it takes many years before it affects the heart. They are setting up the testing to get failure by any means necessary. That's what happens when you have an interest in Big Pharma solutions with financial and other more insidious agendas..
They are setting up the testing to get failure by any means necessary.
^ This.
That's why the 2 or 3 clinical studies that everyone found so "disappointing" had the results they had. They 1) waited too late in the disease process to start and 2) almost deliberately designed the study to test an effect that we know HCQ can't possibly achieve.
The theory is that if it acts as a zinc ionophore inhibiting viral replication (which we have evidence of) or changes the pH internally to the cell, both of these must be achieved prior to infection. That everyone seems to have a fetish for testing HCQ on already sick patients admitted to the ICU suggests that either the researchers are stupid (unlikely) or deliberately modifying the study to obtain the results they want (likely).
It prolongs repolarization of the heart, prolonging the QT interval. If the next heart beats happenings while the heart is still repolarizing this is considered an R on T phenomenon and leads to torsades de point and cardiac arrest.
It doesn't happen often, most are fine, but given that we are talking about sudden cardiac death, this is not a benign issue.
Some have underlying heart disease or take other meds that can be additive
You can also track this with an EKG, if repoladizafion is brjsk enough then there's no issues (qt interval )
Even if you do have heart issues, it takes many years before it affects the heart. They are setting up the testing to get failure by any means necessary. That's what happens when you have an interest in Big Pharma solutions with financial and other more insidious agendas..
^ This.
That's why the 2 or 3 clinical studies that everyone found so "disappointing" had the results they had. They 1) waited too late in the disease process to start and 2) almost deliberately designed the study to test an effect that we know HCQ can't possibly achieve.
The theory is that if it acts as a zinc ionophore inhibiting viral replication (which we have evidence of) or changes the pH internally to the cell, both of these must be achieved prior to infection. That everyone seems to have a fetish for testing HCQ on already sick patients admitted to the ICU suggests that either the researchers are stupid (unlikely) or deliberately modifying the study to obtain the results they want (likely).
It prolongs repolarization of the heart, prolonging the QT interval. If the next heart beats happenings while the heart is still repolarizing this is considered an R on T phenomenon and leads to torsades de point and cardiac arrest.
It doesn't happen often, most are fine, but given that we are talking about sudden cardiac death, this is not a benign issue.
Some have underlying heart disease or take other meds that can be additive
You can also track this with an EKG, if repoladizafion is brjsk enough then there's no issues (qt interval )